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lous atrophy, doctors consider the biop- greater likelihood that gluten sensitivity antibodies. Results have been very im-
sy negative. Early inflammatory changes is present and that celiac dis-ease may pressive thus far. Salivary antibody test-
such as the infiltration of lymphocytes develop. The bottom line is, if you have ing has also been employed as a result of
(a localized inflammatory response) are one or both genes and you have symp- animal studies which showed that when
ignored when they could be viewed as toms or dis-ease(s) suggesting the possi- an antigen (gluten) was taken in orally,
a red flag that something is not right. In bility of celiac it is worth doing a trial of antibodies were not detected in blood
conventional medicine the tendency is gluten elimination from your diet for a but were present in the animal’s stool
to wait until something is completely minimum of 2-3 weeks to see its impact. and saliva.
broken before trying to fix it, instead of Some researchers believe that the pres- As with any dis-ease state there are
looking at signs and symptoms as signals ence of the HLA DQ1 and DQ3 genes often many pieces to the puzzle so if go-
that something is not right and needs to also increase the likelihood that gluten ing gluten free does not immediately re-
be addressed. Also, the earlier in the dis- is a problem for that patient. solve or improve your symptoms or dis-
ease process that we begin to intervene, Recently researchers have been ex- ease state there are a couple of things
the better chance we have of improving ploring the use of stool testing as a non- to consider. Because it can take years to
and possibly reversing the dis-ease. invasive way to detect gluten sensitivity develop symptoms or dis-ease, it may
Blood tests which are relied upon long before celiac develops. Since it is take several months or more to improve
for diagnosis include the presence of in the intestines that the offending food, or resolve them. I always remind my
antigliadin and tissue transglutaminase in this case, gluten, has direct contact patients that they are called patient for
IgA antibodies beyond a certain level. with the microvilli, it would make sense a reason. Patience is an important at-
I believe that if a patient is producing that if the reaction is occurring in the in- tribute, especially when we are talking
these antibodies at any level it suggests testines the stool might be the best place about healing naturally. When we pro-
that the body is reacting to gluten. No to check for the presence of antigliadin vide the body with what it needs; proper
test is one hundred percent accurate
so we must take a patient’s symptoms
into account as well as the laboratory
data when evaluating test results. Also,
because of the way that the gliadin test
is performed in conventional laborato-
ries, about thirty gliadin antibodies are
not picked up. Therefore, many cases of
gluten sensitivity and/or celiac are being
missed.
There is genetic testing with HLA
DQ2 and DQ8 typing which can some-
times be useful in determining the prob-
ability that one has or may develop glu-
ten sensitivity or celiac dis-ease. In the
setting of positive blood tests the pres-
ence of these genes imparts an even
October 2009
19
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