have a positive effect on our cells. By not eating for a certain interval, healthy cells slow their metabolism and enter a repair and “hibernation” mode until fed again. This decreases the risk of any toxic effects that can come with chemotherapy and radiation therapy. Research has found that starvation-dependent stress by means of a fasting-based intervention is capable of dif- ferentially protecting normal versus cancer cells against high-dose chemotherapy in cell culture.
A more recent study found that fasting could slow the growth of tumor cells to
improve the effectiveness of chemotherapy. There is even evidence that fasting after chemotherapy can help to reduce the risk of DNA damage from the treatment itself. Therefore, cancer cells might become more sensitive to chemotherapy treatment while engaging in intervals of fasting. In a few small-scale case studies, fasting was well- tolerated and associated with a self-report- ed reduction in multiple chemotherapy- induced side effects. Due to these findings, researchers have concluded that fasting cycles in combination with chemotherapy could extend the survival of cancer patients by both retarding tumor progression and reducing adverse effects.
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Updates in IV Nutrients Intravenous (IV) Vitamin C (ascorbic acid) has been gaining popularity in the oncology world. Vitamin C for cancer care was first documented by Ewan Cameron and Allan Campbell in the 1970s. Their work got the attention of Nobel Prize winning Linus Pauling who further ex- panded the use and popularity of vitamin C in cancer care. Initial studies were on oral doses of vitamin C, however, blood concentrations achieved were less than 300 µM, not enough to be considered a pharmacologic dose, compared to blood concentrations upwards of 20 mM achieved via IV administration. Additionally, due to bowel tolerance (diarrhea) side effects of oral vitamin C dosing, intravenous vitamin C is a preferred route. Several studies have shown that when plasma concentrations of ascorbic acid are at pharmacological dose (0.1-100 mM) cell division decreases in a variety of cancer cells, specifically, in pros- tate, pancreatic, liver, colon, certain lung cancers and nervous system cancers.
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Studies show combination of Vitamin C with other antioxidants like N-Acet- ylcysteine, improve the antitumor effect by reducing VEGF, vascular endothelial growth factor, a polypeptide that increases the development of new blood vessels to areas of rapidly dividing cells. A trial evaluating the combination of IV vitamin C and chemotherapy agents carboplatin and paclitaxel included twenty seven women with advanced stage ovarian cancer. (Stage 3 and 4). The patients were randomly divided into two groups, one to receive chemotherapy alone and the other chemo- therapy with IV vitamin C. Chemotherapy was given for 6 months at same intervals for both groups. IV vitamin C was given for a total of 12 months. Results revealed a reduction in toxic side effects in the patients who additionally received IV Vitamin C. IV vitamin C was shown to improve quality of life during chemo and or radiation therapy in breast cancer patients. IV vitamin C used as solo therapy in terminal cancer improved quality of life and reduced cancer-related side effects.
IV vitamin C is a very tolerated treat- ment with limited side effects. Research has shown vitamin C can be safely used in pharmacological doses in individuals with normal kidney function. Those with kidney disease or compromise or who have the in- herited disorder known as G6PD deficiency would not be candidates for IV vitamin C therapy due to risk of toxicity. Vitamin C can increase the absorption of iron and
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