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PROMOTION Further reading


◆ Alster TS, Tehrani M. Treatment of cellulite with optical devices: an overview with practical considerations. Lasers Surg Med. 2006;38:727–30.


◆ Bereiter-Hahn J, Blasé C. Ultrasonic characterisation of biological cells. In: Kundu T, editor. Ultrasonic Nondestructive Evaluation: Engeneering and Biological Material Characterisation. Boca Raton: CRC Pr; 2003. pp. 725–59.


Before (left) and after (right) treatment with HERST therapy


cellulite degree I (Figure 4). Hyperaemia was clearly visible


with LCCT at the site of HERST treatment, starting immediately thereafter and lasting for a number of days. In the pre-treated thigh, it is possible to see areas with a low


thigh, it is po“In the pre-treated ” ssible to


see areas with a low perfusion (black in colour) typical of


macro-nodular cellulite degree III.


perfusion (black in colour) typical of macro-nodular cellulite degree III (Figure 5). In the post-treated thigh, one can clearly see an improvement to the area without nodules and


increased homogenisation of light colours corresponding with well-perfused areas, all typical proof of cellulite degree I (Figure 6).


Conclusions The encouraging results obtained in this study reveal that HERST is an interesting non-invasive therapy for cellulite, not only by strengthening the skin’s scaffolding fabric16


, but also


by remodelling the subcutaneous fat tissue. Further studies should investigate whether parameters such as the patient’s age (adolescent, adult or elderly females), body- composition (obesity), and the stage of cellulite have an influence on the outcomes of HERST.


◆ Brodmann M, Ramschak H, Schreiber F, et al. Venous thrombosis after extracorporeal shock-wave lithotripsy in a patient with heterozygous APC-resistance. Thromb Haemost. 1998;80:861.


◆ Delius M, Draenert K, Al Diek Y, et al. Biological effect of shock waves: In vivo effect of high energy pulses on rabbit bone. Ultrasound Med Biol. 1995;21:1219–25.


◆ Gerdersmeyer L, Maier M, Haake M, et al. Physikalisch-technische Grundlagen der extrakorporalen Stoßwellentherapie (ESWT) Der Orthopäde. 2002;31:610–17.


◆ Gerdesmeyer L, von Eiff C, Horn C, et al. Antibacterial effects of extracorporeal shock waves. Ultrasound in Med and Biol. 2005;31:115–19.


◆ Haeussler E, Kiefer W. Anregung von Stoßwellen in Flüssigkeiten durch Hochgeschwindigkeits- Wassertropfen. Verhandlungen Dtsch Phys Gesellschaft (VI) 1971;6:786–9.


◆ Neuland H, Kesselman-Evans Z, Duchstein H-J, et al. Outline of the Molecular and biological effects of the Extracorporal Shockwaves (ESW) on the Human Organism. Orthopädische Praxis. 2004;9:488–92.


◆ Nishida T, Shimokawa H, Oi K, et al. Extracorporeal cardiac shock wave therapy markedly ameliorates ischemia-induced myocardial dysfunction in pigs in vivo. Circulation. 2004;110:3055–61.


 Figures ç Dr Pablo Naranjo Garc” a


◆ Sapozhnikov OA, Khokhlova VA, Baileya MR, et al. Effect of overpressure and pulse repetition frequency on cavitation in shock wave lithotripsy. J Acoust Soc Am. 2002;112:1183–95.


References


1. Wess O. Physics and technology of shock wave and pressure wave therapy. 9th International Congress of the International Society for Musculoskeletal Shockwave Therapy (ISMST) News Letter ISMST. 2006;2:2–12.


2. Roupe et al 1997 3. Urhahne 2005


4. Siems W, Grune T, Voss P, et al. Anti-fibrosclerotic effects of shock wave therapy in lipedema and cellulite. BioFactors. 2005;24:275–82.


5. Wang et al 2006


6. Koshiyama K, Kodama T, Yano T, et al. Structural change in lipid bilayers and water penetration induced by shock waves: Molecular dynamics simulations. Biophys J. 2006;91:2198Ð 205.


60 ❚ 7. Moosavi-Nejad 2006


8. Kato K, Fujimura M, Nakagawa A, et al. Pressure-dependent effect of shock waves on rat brain: induction of neuronal apoptosis mediated by a caspase-dependent pathway. J Neurosurg. 2007;106:667–76.


9. M• ller G, N• rnberger F. Anatomical principles of the so-called


Ò celluliteÓ Arch Dematol Forsch. 1972;244:171–2.


10. N• rnberger F, M• ller G. So-called cellulite: an invented disease. J Dermatol Surg Oncol. 1978;4:221–9.


11. Quatresooz P, Xhauflaire-Uhoda E, Pi•r ard-Franchimont C, et al. Cellulite histopathology and related mechanobiology. Int J Cosmetic Sci. 2006;28:207–10.


12. Tikjob et al 1984


13. Mole B, Blanchemaison P, Elia D, et al. High frequency ultrasonography and celluscore: an improvement in the objective evaluation of cellulite phenomenon. Annales de chirurgie plastique esth•t ique. 2004;49:387–95.


14. Hoffman et al 1989


15. Angehrn F, Kuhn C, Voss A. Can cellulite be treated with low-energy extracorporeal shock wave therapy? Clin Interv Aging. 2007;2:623–30.


16. Kippenberger S, Loitsch S, Guschel M, et al. Mechanical stretch stimulates PKB/Akt phosphorylation in epidermal cells via angiotensin II type 1 receptor and epidermal growth factor receptor. J Biol Chem. 2005;280:3060–7.


◆ Schaden W, Thiele R, Kölpl C, et al. Extracorporeal shock wave therapy (ESWT) in skin lesions. 9th International Congress of the International Society for Musculoskeletal Shockwave Therapy (ISMST) News Letter ISMST. 2006;2:13–14.


◆ Wilbert DM. A comparative review of extracorporeal shock wave generation. BJU Int. 2002;90:507–11.


◆ Wolfrum B, Ohl C-D, Mettin R, et al. 2003. Die Bedeutung von Kavitationsblasen für transiente Membranpermeabilisierung und Zellschädigung Fortschritte der Akustik—DAGA 2003Aachen, 826–7.7M. Vorländer, Deutsche Gesellschaft für Akustik e.V. (DEGA) Oldenburg.


June 2012 | prime-journal.com


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