Like Alzheimer’s, studies indicate
that Parkinson’s disease (PD) results from mitochondrial dysfunction and oxidative damage that triggers the production of an abnormal, inflammatory protein. In PD, the protein is called alpha-synuclein.
Clinical studies show that high doses of CoQ10 (1,200 mg/day) hinders the development of PD disability and appears to slow the otherwise inevitable decline of patients with this disease. (J. Bioenerg. and Biomemb. 2004 August; JAMA Neurol- ogy 2002 October). Noting that CoQ10 is generally “a promising agent for neuropro- tection in neurodegenerative diseases,” re- searchers in a later study tested ubiquinone, ubiquinol, and CoQ10 in two food formula- tions. They concluded that the study results “provide further evidence that administra- tion of CoQ10 is a promising therapeutic strategy for the treatment of PD.” (J Neuro- chem. 2008 Mar).
Cardiovascular Disease (CVD)/ Statin Drug Depletion
CoQ10 benefits the heart by (1) main- taining the normal oxidative state of LDL cholesterol (Mol. Mech. Health & Dis., CRC Press, 2001), (2) supporting circulatory health (a 2007 meta-analysis of 12 clinical trials, including 362 hypertensive patients, found that 100-120mg of supplemental CoQ10 reduces blood pressure) (J Hum Hypertens. 2007 Apr), (3) promoting opti- mal heart muscle function, and (4) possibly improving the health of blood vessel walls (Biochem Biophys Res Commun. 2002).
Conventional physicians primarily recommend statin drugs (Lipitor, Crestor, Zocor, Simvastatin) to reduce the risk of cardiovascular events and heart disease. According to numerous integrative heart disease experts (including Julian Whita- ker, MD, Stephen Sinatra, MD, and Joseph Mercola, DO), however, these drugs can actually increase risk of heart failure by depleting the body of CoQ10. Statins lower CoQ10 levels by (1) reducing the blood cholesterol that transports CoQ10 and other fat-soluble antioxidants; and (2) block- ing the pathway that is involved in both cholesterol and CoQ10 production. The loss of CoQ10 reduces cellular energy and increases free radicals which, in turn, can further damage mitochondrial DNA that sets off a health-destroying cycle of more of the same. In a double-blind, placebo controlled study of subjects taking statins who reported mild-to-moderate muscle pain, 50mg of CoQ10 administered twice daily for 30 days reduced the pain intensity
by 33.1% and interference with daily activi- ties by 40.3%, and symptoms improved in 75% of the subjects given CoQ10. (Medical Science Monitor 2014).
Depression Mitochondrial dysfunction and elevated
brain oxidative-stress levels are character- istic of major depression, bipolar disorder, and schizophrenia. Sufferers thus have low- er CoQ10 and cellular antioxidant levels, and higher markers of oxidant damage, than healthy controls. In one study, 1,200mg/ day of CoQ10 significantly reduced depres- sion symptom severity during treatment of older adults with bipolar disorder. (J Geriatr Psychiatry Neurol. 2012).
Diabetes & Metabolic Syndrome CoQ10 deficiency is closely correlated with reduced long-term blood sugar control and many of the complications of diabe- tes, including diabetic neuropathy (nerve damage), nephropathy (kidney damage), and endothelial dysfunction (an imbalance related to the inner lining of blood vessels) that causes cardiovascular damage. (J Phar- macol Sci. 2008 Jun; Methods Find Exp Clin Pharmacol. 2008 Jun).
One study proved that adding CoQ10
to a healthy Mediterranean diet further reduces oxidant stress and fat oxidation in the period immediately following a meal (postprandial), when the body is especially vulnerable to damage [Age (Dordr). 2011 Dec]. In other studies, supplementation with 200 mg/day of ubiquinol reduced levels of hemoglobin A1c, a marker of blood sugar
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